Platysma muscle, innervated by the facial nerve, can be involved in synkinesis as well, but thus far has been little investigated. Botox. However, after 3-5 months, when the effect of the toxin wears off, your synkinesis will return. Botulinum Neurotoxin (Botox) Injections The standard medical management for HFS is botulinum neurotoxin (BoNT) injections , which provides low-risk but limited symptomatic relief. Botox, when injected into isolated spastic muscles reduce the synkinesis symptoms allowing for retraining of the brain and muscles while the Botox blocks the nerve/muscle actions. Objectives/hypothesis: Facial synkinesis and hyperkinesis commonly impair the outcome of facial nerve palsy. The duration of this process is typically 10 to 20 seconds. Synkinesis refers to involuntary movements of the face - usually as a result of incomplete recovery from facial nerve damage (facial paralysis). Botox is often an effective treatment for synkinesis because it has the ability to reduce activity of the overactive and uncoordinated muscles. Physical Therapy is typically coupled with the Botox treatments for ultimate results and the ongoing retraining process. The aim of our study is to evaluate the presence of platysma synkinesis and its clinical evolution after onabotulinumtoxinA (BoNT-A) (Botox(®) ; Allergan Pharmaceuticals, Irvine, CA) injections. Initially used for reducing hyperkinesis after facial palsy, [23] Botox was later attempted on patients with post-facial palsy synkinesis to reduce unwanted movements. Botox works by blocking release of a neurotransmitter called acetylcholine, which is what nerves use to tell muscles to contract, or tighten. Facial synkinesis is the unwanted movement of certain facial muscles following facial paralysis. Hence, Botulinum toxin relaxes muscles which are too tight and can prevent involuntary facial movements, or synkinesis. Botulinum toxin type A has shown positive results in the treatment of these symptoms. But its role is most commonly done in the superficial face and platysmal neck muscles that are innervated by the seventh cranial nerve. After the synkinesis is inhibited, the patient relaxes the primary movement (D). Periodic injections of Botox (botulinum toxin) may offer some temporary relief and make synkinesis less pronounced. Botox can be a great way to treat the symptoms of synkinesis. Botox for facial synkinesis is an established treatment with proven effectiveness. I help patients like you all the time who have residual side effects from a previous Bell’s palsy diagnosis. Botulinum Toxin (Botox) Injection for Reducing Synkinesis Our experience is reported in this article. Botox (botulinum toxin) is a new and versatile tool for the treatment of synkinesis. As synkinesis becomes apparent (B), the patient maintains activity of the primary mover while releasing the synkinesis (C). Effective for dates of service on or after October 15, 2010, the following indication has been added for BOTOX® (onabotulinumtoxinA): the prophylaxis of headaches in adult patients with Chronic Migraine (= 15 days per month with headache lasting 4 hours a day or longer. Symptoms include: abnormal muscle contractions of the eye, mouth, mid-face and neck, tightness, …
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